3 plasmids (pGL1124, pGL1224 and pGL1217) had been built to allow the replacement of 1 allele of CYC9
showed a phenotype with improved nodules numbers and contaminated cell density and dimensions. Moreover, these overexpressed nodules set much more nitrogen and also the existence of vital nitroge
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As well as in vivo antileishmanial efficacy of a mixture therapy of diminazene and artesunate towards L
COX-2/PGE2 pathway is elevated in murine and human diabetic monocytes/macrophages. Unsuitable PGE2 activity may manage the inflammatory phenotype of wound macrophages, which is not conducive on the repair of diabetic wounds 37. The duality of PGE2 and the complexity of the body involve us to investi
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